TY - JOUR
T1 - 14th International Workshop on Ataxia-Telangiectasia ATW2012
AU - Pandita, Tej K.
N1 - Funding Information:
Meeting support was provided by the NIH, NINDS, USA; Departments of Biotechnology, Science and Technology; CSIR, ICMR, Board of Radiation and Nuclear Sciences, Department of Atomic Energy, Defense Research and Development Organization, Government of India; Association Pour la Recherche sur l’Ataxie-Telangiectasie (APRAT); BrAshA-T Australia; AT Children's Project, Florida, USA; AT Society, Harpenden, UK; Asociacion Espnola Familia Ataxia Telangiectasia; Varian Medical Systems, USA; Abcam Inc., USA and Delhi University, India. Thanks are due to the local organizers, Drs. B.C. Das, B.S. Dwarakanath, D. Saluja and V. Brahmachari. Thanks are also due to T. Paull, K.K. Khanna, D. Cortez, P. McKinnon, J. Tyler, S. Mitra, C. Hunt and N. Horikoshi for their contribution to preparing this meeting report.
PY - 2012/10/1
Y1 - 2012/10/1
N2 - The fourteenth international Ataxia-Telangiectasia Workshop 2012 (ATW2012) (www.atw2012.com) on ataxia-telangiectasia (AT) and the role of the ataxia telangiectasia mutated (ATM) gene in DNA repair, neurological disease, cancer and related topics was held from February 07 to 11, 2012 in Delhi, India. The international ATW2012 meeting reported the latest advances in ATM research as well as potential therapeutic treatments for A-T. The meeting was attended by a productive mix of scientists, ranging from those prominent in the initial characterization of the underlying genetic defect to young scientists just entering the field. In broad terms, three main themes were discussed at the meeting: first, a wealth of new details emerged on DNA damage signaling/repair mechanisms for which ATM is a critical element; secondly, important functions for ATM in previously unrelated cellular pathways were identified; and thirdly, new physiological effects and potential therapeutic treatments related to A-T were presented. This report summarizes below a sampling of the many interesting results from the meeting.
AB - The fourteenth international Ataxia-Telangiectasia Workshop 2012 (ATW2012) (www.atw2012.com) on ataxia-telangiectasia (AT) and the role of the ataxia telangiectasia mutated (ATM) gene in DNA repair, neurological disease, cancer and related topics was held from February 07 to 11, 2012 in Delhi, India. The international ATW2012 meeting reported the latest advances in ATM research as well as potential therapeutic treatments for A-T. The meeting was attended by a productive mix of scientists, ranging from those prominent in the initial characterization of the underlying genetic defect to young scientists just entering the field. In broad terms, three main themes were discussed at the meeting: first, a wealth of new details emerged on DNA damage signaling/repair mechanisms for which ATM is a critical element; secondly, important functions for ATM in previously unrelated cellular pathways were identified; and thirdly, new physiological effects and potential therapeutic treatments related to A-T were presented. This report summarizes below a sampling of the many interesting results from the meeting.
KW - ATM
KW - Cancer
KW - Chromatin structure
KW - DNA repair
KW - Neurological diseases
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U2 - 10.1016/j.dnarep.2012.07.003
DO - 10.1016/j.dnarep.2012.07.003
M3 - Article
C2 - 22921572
AN - SCOPUS:84867215454
SN - 1568-7864
VL - 11
SP - 853
EP - 856
JO - DNA Repair
JF - DNA Repair
IS - 10
ER -