Correction to: Newborn Mice Lacking the Gene for Cyp1a1 Are More Susceptible to Oxygen-Mediated Lung Injury, and Are Rescued by Postnatal β-Naphthoflavone Administration: Implications for Bronchopulmonary Dysplasia in Premature Infants

Paramahamsa Maturu; Yanhong Wei-Liang; Weiwu Jiang; Lihua Wang; Krithika Lingappan; Roberto Barrios; Yao Liang; Bhagavatula Moorthy; Xanthi I. Couroucli

doi:10.1093/toxsci/kfac031

Correction to: Newborn Mice Lacking the Gene for Cyp1a1 Are More Susceptible to Oxygen-Mediated Lung Injury, and Are Rescued by Postnatal β-Naphthoflavone Administration: Implications for Bronchopulmonary Dysplasia in Premature Infants

Paramahamsa Maturu, Yanhong Wei-Liang, Weiwu Jiang, Lihua Wang, Krithika Lingappan, Roberto Barrios, Yao Liang, Bhagavatula Moorthy, Xanthi I. Couroucli

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Abstract

Toxicological Sciences, Volume 157, Issue 1, May 2017, Pages 260 271, https://doi.org/10.1093/toxsci/kfx036 In the originally published version of this manuscript, two of the representative pathology slides (3F and 3H) in Figure 3 are overly similar. An updated Figure 3, with 3H replaced with a different pathology slide, is published below. These details have been corrected only in this correction notice to preserve the published version of record. (Figure Presented).

Original language	English (US)
Pages (from-to)	187
Number of pages	1
Journal	Toxicological Sciences
Volume	187
Issue number	1
DOIs	https://doi.org/10.1093/toxsci/kfac031
State	Published - May 1 2022

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Maturu, P., Wei-Liang, Y., Jiang, W., Wang, L., Lingappan, K., Barrios, R., Liang, Y., Moorthy, B., & Couroucli, X. I. (2022). Correction to: Newborn Mice Lacking the Gene for Cyp1a1 Are More Susceptible to Oxygen-Mediated Lung Injury, and Are Rescued by Postnatal β-Naphthoflavone Administration: Implications for Bronchopulmonary Dysplasia in Premature Infants. Toxicological Sciences, 187(1), 187. https://doi.org/10.1093/toxsci/kfac031

Correction to: Newborn Mice Lacking the Gene for Cyp1a1 Are More Susceptible to Oxygen-Mediated Lung Injury, and Are Rescued by Postnatal β-Naphthoflavone Administration: Implications for Bronchopulmonary Dysplasia in Premature Infants. / Maturu, Paramahamsa; Wei-Liang, Yanhong; Jiang, Weiwu et al.
In: Toxicological Sciences, Vol. 187, No. 1, 01.05.2022, p. 187.

Research output: Contribution to journal › Article › peer-review

Maturu, P, Wei-Liang, Y, Jiang, W, Wang, L, Lingappan, K, Barrios, R, Liang, Y, Moorthy, B & Couroucli, XI 2022, 'Correction to: Newborn Mice Lacking the Gene for Cyp1a1 Are More Susceptible to Oxygen-Mediated Lung Injury, and Are Rescued by Postnatal β-Naphthoflavone Administration: Implications for Bronchopulmonary Dysplasia in Premature Infants', Toxicological Sciences, vol. 187, no. 1, pp. 187. https://doi.org/10.1093/toxsci/kfac031

Maturu P, Wei-Liang Y, Jiang W, Wang L, Lingappan K, Barrios R et al. Correction to: Newborn Mice Lacking the Gene for Cyp1a1 Are More Susceptible to Oxygen-Mediated Lung Injury, and Are Rescued by Postnatal β-Naphthoflavone Administration: Implications for Bronchopulmonary Dysplasia in Premature Infants. Toxicological Sciences. 2022 May 1;187(1):187. doi: 10.1093/toxsci/kfac031

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title = "Correction to: Newborn Mice Lacking the Gene for Cyp1a1 Are More Susceptible to Oxygen-Mediated Lung Injury, and Are Rescued by Postnatal β-Naphthoflavone Administration: Implications for Bronchopulmonary Dysplasia in Premature Infants",

abstract = "Toxicological Sciences, Volume 157, Issue 1, May 2017, Pages 260 271, https://doi.org/10.1093/toxsci/kfx036 In the originally published version of this manuscript, two of the representative pathology slides (3F and 3H) in Figure 3 are overly similar. An updated Figure 3, with 3H replaced with a different pathology slide, is published below. These details have been corrected only in this correction notice to preserve the published version of record. (Figure Presented).",

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AU - Barrios, Roberto

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Correction to: Newborn Mice Lacking the Gene for Cyp1a1 Are More Susceptible to Oxygen-Mediated Lung Injury, and Are Rescued by Postnatal β-Naphthoflavone Administration: Implications for Bronchopulmonary Dysplasia in Premature Infants

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